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The Mystery "Virus"
When in doubt, blame the invisible boogeymen.
One of the beauties of the germ theory of disease is that there are many escape clauses that have been built into the theory that allow for the lie to persist in the face of contradictory evidence. For example, if the researchers find the “virus” or bacteria within a host without disease, the person must be an asymptomatic carrier. If they are unable to find the specific “virus” or bacteria in someone with the symptoms of disease associated with it, that person must have one of the many other “viruses” or bacteria that cause the exact same symptoms. If the researchers attempt to infect someone with the “virus” but no illness occurs, then the person must have been exposed at some earlier time and built up antibodies and immunity. If the same “viral” particles are found in electron microspe images of fluids and tissues where they shouldn't be, then they are not the “virus” but must be some other “virus-like” particles or normal cellular constituents. All of these examples allow for the “viral” hypothesis to exist in face of contradictory evidence that should falsify the hypothesis.
In the spirit of continuing to expose these ridiculous tricks virology uses to keep the pseudoscientific wheel turning, I want to focus on another escape clause that is seemingly becoming more and more common these days. This is a concept known as the mystery “virus.” This excuse is thrown out there in order to provide a cause for unexplained symptoms of disease when the usual known “viral” culprits are not detected by the fraudulent tests. We have regularly seen a variation of this in practice when doctors declare “it's probably viral and will run its course” when they can not come up with a diagnosis based on the non-specific symptoms and inaccurate tests. We saw this happen this past year when people tested negative for “Covid,” influenza, and RSV and yet, they were still suffering from the exact same illness. These cases of the same symptoms are all lumped together under the umbrella term “influenza-like illness.” Apparently, it is not important to determine what the unknown cause of these symptoms is if it's just the same old symptoms we see all of the time. It is chalked up to “it's just a respiratory virus.” This makes the hysteria over the same old symptoms that sounded the alarm for researchers to look for “SARS-COV-2” seem rather illogical. Virology has made it so that it is not necessary to test positive for any of the “viruses” in order for one to be diagnosed with a “virus.” Doctors such as Dr. Roy Gulick, chief of infectious disease at New York-Presbyterian and Weill Cornell Medicine, will just tell you that you don't need to know what “virus” you have. Go home and it will go away:
People are getting sick with mystery illnesses and testing negative for COVID, RSV, and flu. Here's why.
“General practitioner Stephanie de Giorgio from the UK said, similarly, that some kind of "not-flu, not-covid, not-RSV thing" was going around her workplace, and "felt bloody awful," prompting a fever and sore throat.”
“This red-hot level of "influenza-like illness" is a barometer that's based on patient symptoms (not viral tests) so it likely encompasses many cases of flu, COVID, and several other respiratory diseases showing up on doctors' doorsteps.
Trying to tell whether you've got the flu or COVID based on your symptoms? Good luck.
"Fever, muscle aches, cough, headache, those are going to be common," Dr. Roy Gulick, chief of infectious disease at New York-Presbyterian and Weill Cornell Medicine, told Insider. "You really can't tell the difference between flu and COVID."
"Most people probably don't need to know what they have," Gulick said. "They're gonna just go through the illness, and then get better with supportive care." Antibiotics are of no help for viral illnesses, so plenty of bedrest, fluids, and Tylenol or ibuprofen for fevers and aches is the tried and true method for at-home care.
Another interesting way that this escape clause has been used in the past is to explain mysterious vaccine ingredients. In the case of the smallpox vaccine, for over a century it was assumed that the cowpox “virus” was the “virus” utilized to make the vaccine. However, it is now claimed that molecular and genetic testing dispelled this myth and that it is, in fact, a mystery “virus” no one has ever identified that “protects” people from these diseases. No one knows how, when, where, or why this “virus” entered the smallpox vaccine. They don't know where it originated, what its natural host is, or if it even persists in the wild. All they know is that they know nothing about it, and fittingly, this ghost “virus” has been given the moniker vaccinia:
The mystery virus that protects against monkeypox
“For more than a century, the smallpox vaccine was widely assumed by the scientific community to be made from cowpox – this is the explanation still found in many websites and curriculums worldwide. But in 1939, nearly 150 years after vaccination was invented, molecular tests revealed that it's not. More recently, genetic sequencing has confirmed these findings. Instead, the vaccines that were used to eradicate smallpox, and those in use today against monkeypox, are based on an unknown virus that no one has been able to identify – a "ghost" pathogen that has only ever been found in vaccine form.
Despite an 83-year search, no one knows how, why or precisely when this imposter appeared in the smallpox vaccine, or whether it still exists in the wild. Only one thing is clear: millions who lived through the reign of smallpox owe their lives to its existence. Without it, the current monkeypox outbreak is likely to have spread even more rapidly.
"For many years, until 1939, people assumed that what we call vaccinia, the smallpox vaccine, was the same as cowpox," says José Esparza, a virologist and fellow of the Robert Koch Institute, Germany. "And then it was discovered that they were different. And since then, we accept that cowpox is one virus, and vaccinia is another virus of unknown origin.
How has this happened? Where could this virus have come from? And will we ever be able to find it in its natural host?”
In 2021, there was an outbreak of a mystery “virus” in Pakistan where the victims were said to be suffering from the exact same symptoms as Dengue fever. These people were experiencing reduced platelets and lowered white blood cells during a time when Dengue was rampant, but they consistently tested negative for the Dengue “virus.” It was suspected of being a “virus” from the same family as Dengue as the people suffered the exact same non-specific symptoms associated with the disease and were provided the exact same treatments. However, no culprit was ever identified and the exact same symptoms were blamed once again on a mystery “virus” rather than exploring alternative possibilities:
Mystery virus outbreak that depletes white blood cells baffles scientists as cases soar
“The mystery illness has been diagnosed across a number of different hospitals in Karachi, with patients showing symptoms including reduced platelets and white blood cells. Pakistan is already experiencing an epidemic of dengue fever, with the latest outbreak infecting more than 16,000 people acording to the World Health Organization (WHO). However, patients in Karachi have been testing negative for the mosquito-borne disease despite showing the typical symptoms.
According to a report in The News, health experts suspect a pathogen from the arbovirus family of viruses could be responsible.
Professor Saeed Khan, head of molecular pathology at the Dow University of Health Sciences in Karachi, said: "For a couple of weeks, we are seeing cases of viral fever, in which platelets and white blood cells and dropping while other clinical symptoms are also similar to the dengue fever.
"But when NS1 antigen of these patients is performed, their tests come out to be negative."
Although the patients have tested negative for dengue fever, they reportedly required the same protocol treatments.”
The “virus” has always been a distraction to keep people from focusing on more logical explanations for illness and disease. It has served as the perfect scapegoat to keep people from demanding cleaner air, water, food, etc. It has kept people from questioning the negative effects of the fast food consuming and Netflix-binging sedentary lifestyles that they lead on a daily basis. It has kept them from investigating the toxicity of the treatments that have been sold as the cure for their ailments. People look to the “virus” as the explanation for what aflicts them, and when one of the main culprits is absent, they assume that it must be another one of these invisible boogeymen out there that we just haven't identified that is ultimately to blame.
There are many ways that the mystery “virus” label can be utilized to brush off contradictory evidence. As the use of this concept has recently become even more prevalent today in order to seemingly cover up for potential adverse reactions to the “Covid” vaccine as well as other related treatments for these “influenza-like illnesses,” I want to investigate a few recent examples and see if there is fire where we see smoke. Let's explore how this mystery “virus” concept is used to keep this “viral” lie afloat in order to protect the interests of the pharmaceutical companies.
Mystery “Virus” Attacking the Liver
In this first instance, a group of children were stricken with what was claimed to be a mysterious liver disease in the early months of 2022. Upon testing, the usual “viral” culprits were nowhere to be found. Instead of looking for potential causes outside of a “virus,” such as the use of vaccines or other medical treatments in these children, the researchers settled on the idea that it must be a “viral” cause of mysterious origin and went searching. With the help of the latest genomic tricks, the researchers were able to piece together enough sequences claimed to belong to various “viruses.” However, none of the main culprits identified were associated with liver disease. As a non-pathogenic “virus,” known as adeno-associated “virus” 2 (AAV2), was found in the majority of the cases, it was decided that this “harmless virus” was the possible cause. In order to make such a bold claim, the researchers hypothesized that this non-pathogenic “virus” was somehow working with the other “viral” sequences that were detected in order to cause liver disease in children. This became known as the “helper virus” hypothesis:
Rare hepatitis cases in kids linked to 'helper viruses,' studies suggest
“An unusual global outbreak of pediatric hepatitis cases in 2022 could be connected to multiple common viruses, according to multiple studies.
Three studies published March 30 in Nature found that nearly every child with an unexplained hepatitis case had high levels of adeno-associated virus 2, or AAV2. Another virus, adenovirus, may also be tied — though the pair of viruses have never before been associated with human disease. Becker's reported on one of the preprints in late July, a few days after the global count of cases surpassed 1,000.
One of the studies found AAV2 in 13 of 14 young patients with hepatitis, or liver inflammation, and co-infections of "helper viruses" — including Epstein-Barr virus, human herpesvirus 6, and/or enterovirus A71 — were found in 12 of the 14 cases. Another study of a few dozen cases revealed "an association between AAV2 infection and host genetics in disease susceptibility [...] alongside a prominent T-cell infiltrate in liver biopsies."
A third small study conducted in the U.K. yielded similar findings: The abnormal amount of hepatitis cases among children in 2022 seem to be tied to a combination of AAV2 and another aiding virus.”
According to the hypothesis, the non-pathogenic AAV2 started working alongside “helper viruses” such as other “adenoviruses,” herpes “viruses,” Epstein-Barr “viruses,” and/or '“enteroviruses” in order to join together in some kind of Voltron-esque entity so that it could then attack the livers of children.
Of course, this amounts to a game of genetic correlation equalling causation as no “virus” was purified and isolated and proven to cause liver disease. The kids were tested for different “viruses” via PCR and the most common sequences detected were considered the likely suspects, as multiple “viruses” were found within the same individuals at the same time. This led to the unscientific hypothesis that these strings of A,C,T,G’s in a computer database were teaming up and targeting the liver. At least the reporting of CNN showed that this hypothesis was unproven:
Mysterious hepatitis outbreak in kids in 2022 linked to common childhood respiratory virus, studies suggest
“Although the consistent findings across two continents provide strong evidence, several uncertainties remain, including whether AAV2 directly causes liver damage or is "just a bystander."
For example, because they can't replicate on their own, adeno-associated viruses are commonly used as vehicles for gene therapy. Although liver damage has been observed in trials that involve AAV-based approaches, it is rare and generally not fatal, scientists say.
"If AAV2 directly caused hepatitis, one would expect more cases to have been reported," Tacke said.
More studies are needed to understand how AAV2 infection alone, or with another virus, might affect liver cells, he recommended.”
Insterestingly, CNN also pointed out that therapies using “adenovirus” vectors have been linked to liver disease. It just so happens that the “viral” vector being used in the Johnson & Johnson and AstraZeneca “Covid” vaccines is an “adenovirus,” a common type of “virus” that is typically associated with mild cold symptoms when it “infects” someone. This is the same “virus” now being blamed for the mysterious liver disease in children. Here is how this “viral” vector vaccine process is described:
Explaining Johnson & Johnson’s, AstraZeneca’s new COVID-19 vaccines
“Viral vector vaccines use a modified, harmless version of a different virus as a vector, or carrier, to deliver immunity instructions to cells in the body. The body then follows those instructions to build an immune response to the intended virus (in this case, SARS-CoV-2, which causes COVID-19.)
The virus vector being used in the Johnson & Johnson and AstraZeneca vaccines is an adenovirus, a common type of virus that typically causes mild cold symptoms when it infects someone.
COVID-19 viral vector vaccines inject a harmless adenovirus vector, which carries unique genetic information from the COVID-19 virus to human cells. Once it reaches human cells, the vector uses that genetic information with the cell’s machinery to produce a COVID-19 spike protein (a small piece of SARS-CoV-2) on the cell’s surface. That spike protein triggers a response from the body’s immune system to start producing antibodies to COVID-19.”
Two of the “Covid” vaccines on the market are claimed to be created from these harmless “adenoviruses.” It is also known that both the “viral” vector and the mRNA vaccines have been associated with liver damage:
Cases of severe acute liver injury following inactivated SARS-CoV-2 vaccination
“Several cases of severe acute liver injury (ALI) with features of autoimmune hepatitis after SARS-CoV-2 infection or SARS-CoV-2 vaccination have been described.1, 2, 3, 4, 5, 6, 7 Most cases developed ALI following mRNA or viral vector-based vaccines, while only a few reports described the development of ALI after inactivated SARS-CoV-2 vaccines.”
Thus, it is not that far-fetched to believe that the vaccines are potentially a factor in the development of liver disease in children. However, the mainstream media “fact-checkers” were quick to point out that these children were not vaccinated for “Covid” and thus, these vaccines could not have been a factor in this mysterious disease:
Fact Check-No link between hepatitis cases in children and COVID-19 vaccines
“False. There is no link between the COVID-19 vaccine and hepatitis among children in Britain. None of the children had received COVID-19 vaccines. Adenoviruses may be causing the illnesses, but no cause has yet been established for cases in Britain, Europe and the U.S.”
However, “Covid” vaccines are not the only vaccines children receive. There are many other vaccines given in rapid succession to children under the age of 5. Any one of these could be a potential factor in liver disease amongst children. In fact, it was known as far back as 1993, two years after the CDC recommended universal hepatitis B vaccination in children, that those children who were vaccinated against hepatitis B were at an increased risk of liver damage:
Hepatitis B vaccine and liver problems in U.S. children less than 6 years old, 1993 and 1994
“Data to assess the benefits and risks of hepatitis B vaccine for the general population of U.S. children are sparse. This study addressed the problem of external validity found in previous studies of high risk populations by evaluating the benefit of hepatitis B vaccination for the general population of American children. We calculated the risk of liver problems among hepatitis B vaccinated and non-hepatitis B vaccinated children using logistic regression. Hepatitis B vaccinated children had an unadjusted odds ratio of 2.94 and age-adjusted odds ratio of 2.35 for liver problems compared with non-hepatitis B vaccinated children in the 1993 National Health Interview Survey. Hepatitis B vaccinated children had an unadjusted odds ratio of 2.57 and age-adjusted odds ratio of 1.53 for liver problems compared with non-hepatitis B vaccinated children in the 1994 National Health Interview Survey dataset.”
Corroborating research also showed that the Hep B vaccine caused liver damage in mice:
Are Vaccines Damaging Your Baby’s Liver?
“Well here we have research demonstrating a vaccine CAUSING a mitochondrial disorder!
The article’s abstract opens with “Vaccines can have adverse side-effects, and these are predominantly associated with the inclusion of chemical additives such as aluminum hydroxide adjuvant;” and states, “We conclude that exposure of Hepa1-6 cells to a low dose of adjuvanted hepatitis B vaccine leads to loss of mitochondrial integrity, apoptosis induction, and cell death, [emphasis mine] apoptosis effect was observed also in C2C12 mouse myoblast cell line after treated with low dose of vaccine (0.3, 0.1, 0.05 μg/ml). In addition In vivo apoptotic effect of hepatitis B vaccine was observed in mouse liver.”
In other words, the toxicity of the vaccine in very low doses was observed both in actual live mouse liver as well as in cell lines from the mouse. Are we destroying our babies’ livers with these vaccines? Non-alcoholic liver disease has become a virtual epidemic in recent years. Could vaccines be implicated?”
We have to ask ourselves why a hypothetical “helper virus” scenario was created as an explanation for liver disease rather than investigating any of the numerous vaccines injected into children every year that are associated with such disease. According to a June 24th, 2022 NBC report, the amount of children with liver disease was not even at an unusual level at the time the CDC and the WHO alerted the world:
The CDC is still searching for the cause of mysterious liver illness in kids
“A total of 296 potential cases of unexplained hepatitis in young children have been identified so far, the CDC reported Friday. Most of the cases are not new; many were identified in retrospect, with doctors looking as far back as October. And while the number may sound high, they haven’t exceeded the expected yearly number of severe pediatric hepatitis cases.”
What was the point in alerting the world for what was said to be a regular amount of cases of liver disease in children? Perhaps they were anticipating a helping of new cases of liver disease once the “Covid” vaccine was authorized for children under the age of 5? Seven days prior to the NBC News article on the mystery liver disease in children 5 and under, on June 17th, 2022, the FDA authorized the “Covid” vaccines by way of the Emergency Use Authorization (EUA) for use in children 5 and under:
Coronavirus (COVID-19) Update: FDA Authorizes Moderna and Pfizer-BioNTech COVID-19 Vaccines for Children Down to 6 Months of Age
“Today, the U.S. Food and Drug Administration authorized emergency use of the Moderna COVID-19 Vaccine and the Pfizer-BioNTech COVID-19 Vaccine for the prevention of COVID-19 to include use in children down to 6 months of age.”
A day later, the CDC began recommending the vaccines for this age group:
CDC Recommends COVID-19 Vaccines for Young Children
“Today, CDC Director Rochelle P. Walensky, M.D., M.P.H., endorsed the Advisory Committee on Immunization Practices’ (ACIP) recommendation that all children 6 months through 5 years of age should receive a COVID-19 vaccine. This expands eligibility for vaccination to nearly 20 million additional children and means that all Americans ages 6 months and older are now eligible for vaccination.”
Was the “mystery helper virus” hypothesis simply a cover for the potential reactions that children could have encountered from yet another toxic injection added to their already heavily poisoned bodies? Sadly, only time will tell as our children are being unethically experimented on with unproven injections that have already caused a great deal of harm. One thing that is for certain is that there is absolutely no scientific evidence supporting said hypothesis. Belief in this “helper virus” hypothesis has diverted any examination into the more obvious possible contributing factors that should be investigated. As per usual, factors such as adverse vaccine reactions, side effects from toxic pharmaceuticals, poisoning from GMO pesticide-laden foods and sugary drinks, a lack of physical activity and sunlight, etc. are ignored in favor of pseudoscientific data used to blame invisible culprits.
Mystery “Virus” Attacking the Heart
As bad as it is to see our young children injected with these toxins that are contributing to these diseases, older children and young adults are not free from harm either. In fact, the damages from these injections may be even more severe. The risk of myocarditis is already well-known and was added to the vaccine warning labels as a potential reaction. According to the CDC, adolescents and young adult males are singled out as most likely to suffer from this adverse reaction:
Myocarditis and Pericarditis
“Myocarditis and pericarditis have rarely been reported. When reported, the cases have especially been in adolescents and young adult males within several days after mRNA COVID-19 vaccination (Pfizer-BioNTech or Moderna).”
We have recently seen numerous reports of young males and athletes suffering from a condition that most had probably never even heard of before. Sadly, it has become a commonly accepted vaccine reaction and risk. However, even with the known risk, many of these stories of young people succumbing to heart attacks are brushed off as anomalies, such as in the below case of 18-year-old Jordan Brister:
High school senior dies after suffering heart attack at school, officials say
“Officials in Las Vegas say a student died after he unexpectedly suffered cardiac arrest while at school.
According to a post from Amplus Academy, the student was identified as senior Jordan Brister.
A GoFundMe shared by the school said Brister “suddenly and unexpectedly suffered cardiac arrest while at school with no explanation as to why.”
The death of Jordan Brister was eventually blamed on bacterial pneumonia and was labelled a natural death, even though the boy was at school and apparently healthy, with his death being unexpected. Young adults dying from heart attacks is not natural and should not be happening, yet these headlines are becoming all too commonplace now, whether blamed on “viruses,” vaccines, treatments, or natural causes.
While the unfortunate case of Jordan Brister ended up with a defined cause and a death that was ultimately listed as natural (regardless as to how unrealistic that cause was), there are instances where no cause is ever identified and a “virus” is still to blame. This happened recently to a young male named Jacob Brooks, who was experiencing flu-like symptoms and went to the doctor for a check-up. Jacob thought all he had was a typical flu with a cough and explained that he was not experiencing any severe symptoms at the time of his check-up. After initial testing to determine the cause, the young man was told that he needed to be rushed immediately to the hospital. After notifying his loved ones, Jacob was sedated and his condition deteriorated fast. Jacob's plasma was taken out and replaced with fresh plasma. He was connected to a mechanical lung and heart known as an ECMO device. They started dialysis as his kidneys shut down, and his heart had to be shocked back into rhythm seven times. While Jacob was fortunate and ultimately survived his ordeal, the family wanted it to be known that this situation was not related to either “Covid” or the vaccine. They were told that a rare, never named or identified mystery “virus” was the cause:
Mystery virus sends young Houston-area man into organ failure
“MISSOURI CITY, Texas - A family from Missouri City is encouraging others to seek quick treatment for symptoms that don't go away quickly after their son got a virus that shut down his organs.
Jacob Brooks, 22, was an accomplished athlete in high school, and his parents say he has always been healthy and never had underlying conditions. That's why they say they are still in a state of shock that an illness came close to claiming his life. They would like to make it clear that this illness was not COVID-19 or vaccine-related.
"At first, I just thought it was a flu and cough, that's what we went in for. And then doctors were doing their tests, and then they came in saying, ‘we need to get you to the hospital now,’" Jacob says.
That conversation quickly changed to urging the family to get Jacob to a heart center.
"Doctors told me I needed to make some calls, and I was wondering what that meant, so I called and told them I loved them, and then I hung up and that's when they put me down (sedation)," Jacob says.
"The doctor said it was a rare virus that they usually only see between kids 18 to 22 and most of them recover, but every once in a while, you'll have a case like Jacob's, and they don't know why. The virus has never really been identified and never been named. It was so severe, they took all his plasma out of his body and replaced it with new plasma," states Eugene Brooks, Jacob's dad.
Jacob admits he felt bad, which is why he went to the emergency room, but he says he didn't have any heart-related symptoms, so he had no idea he was in heart failure.
"He just deteriorated so fast. They put him on a device called ECMO, and it's basically a mechanical heart and lung. They also put him on dialysis because his kidneys were shutting down. They intubated him, and they automatically just put him to sleep. His heart was shocked, I think seven times, and even during the drive from Houston to San Antonio, they shocked his heart four times, in a matter of three hours during the drive," says Eugene.”
"We are in the position of other parents thinking this could happen to anybody. He was an otherwise healthy 22-year-old, doing what 22-year-olds do and then here we are in a life or death situation. So, that's something I want to just caution all parents: when you hear that cough or that sneeze or that whatever you thought it was, just go get help, even if it's 12 o'clock at night, and you think it's just a cough, maybe it's not," says an encouraging Kim.”
While the mystery “virus” went unnamed, according to Jacob's GoFundMe page, his condition was listed as “Fulminant Viral Myocarditis,” which boils down to severe inflammation of the heart that is said to be caused by a “virus.” It was reiterated that Jacob only had flu-like symptoms and that his mother took him to the Urgent Care when the symptoms persisted into the night. The treatments began and his condition deteriorated:
“We have learned that Jacob suffered from “Fulminant Viral Myocarditis”, which is an uncommon syndrome characterized by sudden and severe diffuse cardiac inflammation. The severity of his condition alone will place him at the very top of the heart transplant list.”
“It began just a few short weeks ago when he complained of flu-like symptoms. When his symptoms continued into the next night, Jacob’s mother decided to take him to the neighborhood Urgent Care. But, what happened next left them in complete shock . . .
Upon initial exam at the Urgent Care, the doctor advised that Jacob be taken immediately by ambulance to a nearby hospital. Within minutes of arrival, the doctor became visibly concerned, as a virus aggressively attacking Jacob’s heart was discovered. Jacob was immediately transported to the Methodist Hospital Heart Center in San Antonio, Texas (about 300 miles away).
Since that time, Jacob has been in the CV ICU where he was intubated and put on life support; including ECMO, Impella, ventilator, and dialysis, and placed into a medically-induced coma. Fulminant Viral Myocarditis syndrome often leads to death resulting from cardiogenic shock, ventricular arrhythmias, or multiorgan system failure. Historically, this syndrome is almost exclusively diagnosed at autopsy.”
As stated before, the family claimed that it was neither “Covid” nor the vaccine that caused Jacob's rapid decline. While we know that “Covid” did not play a part as there is no “SARS-COV-2,” we must take their word on the vaccine as there is no information as to whether Jacob was vaccinated for “Covid” or not. We can only speculate as to what indirect biomarkers were detected upon Jacob's visit that led to his “diagnosis” of a rare mystery “virus.” However, was it the “virus” that caused Jacob's rapid decline or was it the treatment that was implemented based upon the results of whatever testing was done?
As stated by the reports, Jacob was immediately sedated and his condition deteriorated rapidly afterwards. Why did his condition decline so quickly after sedation? Could it have been a response to whatever sedation method was used? This led to the need for a mechanical heart and lung in order to keep Jacob alive. Was the use of this device, combined with sedation and having his plasma replaced, what ultimately led to the need for dialysis once Jacob's kidneys were unable to function due to the unnatural conditions his body was placed under? Jacob was put on life support and a medically induced coma. He had to be shocked back to life seven times, eventually leading to Jacob having to be added to the waiting list for a heart transplant. Was this really the work of a mystery “virus” or was this the result of the various treatments Jacob was subjected to based upon what tests showed at the Urgent Care clinic?
Of course, they will claim that Jacob got there just in the knick of time and that the interventions ultimately saved his life. However, with iatrogenic mistakes being the 3rd (and arguably the 1st) leading cause of death in the US, could it be that the mystery “virus” was a scapegoat to blame the outcome of the hazardous treatments on? Did the doctors actually save Jacob's life or did they nearly take it away, leaving him in a worse condition than he would have been in had he never entered the Urgent Care that fateful night? All we can say for certain is that there was no evidence of any mystery “virus.” It had never even been named or identified.
As for his condition, while there are “viruses” linked to fulminant myocarditis, we can find out from a timely December 2020 literature review, published right after the vaccines became widely used, that the exact etiology of this disease still remains unknown. In one study discussed, only 38% of the cases of fulminant myocarditis were associated with “viral” genomes. Other potential causes include various bacteria, autoimmune conditions such as Lupus, and toxicity from drugs and treatments. In other words, there are numerous factors that they can pull out of their hat and blame for the condition at any given time, including mystery “viruses.” However, the mechanisms for how any of these factors can lead to the severe inflammation of the heart are largely unknown:
Fulminant myocarditis: a comprehensive review from etiology to treatments and outcomes
“While the exact etiology of FM remains largely unknown, our current understanding indicates three main factors contribute to its development. The first factor suggests an infection caused by various pathogens, especially viruses,2,12 which manifest in clinical features observed in FM. In fact, certain types of viral nucleic acids can be detected directly in endomyocardial biopsy (EMB) samples and serum by real-time polymerase chain reaction (PCR) or in situ hybridization (ISH),9,13 such as parvovirus B19 (PVB19), coxsackievirus B3 (CVB3), and cytomegalovirus (CMV).2 Although uncommon, some viruses that usually infect non-cardiac organs, like human immunodeficiency virus (HIV),14 can also induce FM. Importantly, due to the limited sensitivity of detection assays, it is possible to get false-negative results of a viral infection. A multicenter study revealed that only 38% of myocarditis patients can find viral genome in their EMB samples.15 PCR or PCR-based detection techniques show the best performance, reaching sensitivity levels in the range of 50–90%,9,13,16 and an ability to detect multiple viruses in clinical samples.17 In contrast, serological tests and ISH of EMB samples show poorer sensitivity.9 A study reported a poor correlation between PCR and serological results, with only 4% of serological evidence of viral infection being determined by EMB.9 However, PCR-based detection techniques are limited in scope to predicted or suspected viruses due to the need for specific primers for amplification. Recent advances with next-generation sequencing (NGS) has provided clinicians with an ability to obtain unbiased results of possible pathogens of infectious myocarditis.18,19 Interestingly, several uncommon viruses including Epstein Barr virus (41%), human pegivirus (4%), human endogenous retrovirus K (100%), and anellovirus (56%) were found indicating the complexity of the viral constitution in FM. Apart from viruses, bacteria including Chlamydia pneumoniae, Mycobacterium tuberculosis,12 Neisseria meningitidis20 or protozoan such as Plasmodium falciparum, Toxoplasma gondii12 or Giardia lamblia21 have all been reported to trigger FM. Hence, special attention should be paid to infected patients with rapid deterioration of cardiac function.
The second factor contributing to FM development is autoimmune disease. The systemic lupus erythematosus (SLE),22 rheumatism,23 scleroderma24, and Sjogren’s syndrome25 have all been reported to induce FM. By expressing PD-L1 on myocardium, the heart is considered to be well-protected from cardiac-specific T cells, which mostly recognize the α-myosin heavy chain peptide.26 However, a disturbance of the balance of immune homeostasis by autoimmune diseases may enable self-antigens to be exposed to the immune system and mistakenly activate myocardium cytotoxic T cells. This may partially explain the effects of glucocorticoids in treating autoimmune disease-induced FM.27 Since the key pathological process of autoimmune disease-induced FM is self-antigen such as alpha or beta-MHC exposure to the immune system, it is convincing that disturbances of the immune system caused by autoimmune diseases lead to the formation of auto-cytotoxic immune cells, CD3+/CD8+ T cell predominantly, as well as macrophages, targeting the myocardium, eventually leading to FM.28
The third factor contributing to FM development is drug toxicity. Many drugs, especially chemotherapy drugs and certain natural derivatives,29 are toxic to cardiomyocytes,30,31 which is exemplified by increased incidences in check-point inhibitor-related FM.32 Although check-point inhibitors have brought revolutionary advances to the treatment of late-stage malignant cancers they induce the formation of auto-cytotoxic immune cells. These cells subsequently attack the myocardium, resulting in the accumulation of CD3+CD8+ T cells, macrophages, and neutrophils in the heart.33,34 The prevalence rate of cardiac side effects of checkpoint inhibitors is common, which can reach 25% or more.35 Although the prevalence rate of FM from checkpoint inhibitors is <1% and is much lower than other targets, its fatality rate is as high as 40~70%.35,36,37 Therefore, it is important to pay attention to patients receiving chemotherapy who display cardiac function deterioration, as this may be a sign of FM.
To date, there are three major classes of etiological factors in FM known (Table 1). However, the exact mechanism(s) behind each etiological factor remains largely unknown. Importantly, these factors are not completely distinct but have overlapping signaling pathways and cellular responses triggering their effects. It may be hypothesized that the mechanisms underlying FM involve common immune system pathways, for example, check-point inhibitor-induced FM and hypersensitivity induced FM, are related to auto-immune disruption and myocardium cytotoxicity T cell activation.38,39”
While Jacob's parents said that the “Covid” vaccines were not the cause of his condition, it is definitely possible, had he been vaccinated, that this could have played a part in his condition as seen in various case reports released after the introduction of the “Covid” vaccines. In an instance reported in February 2022, a 50-year-old man was diagnosed with fulminant myocarditis after his second mNRA injection. The paper listed fulminant myocarditis as an adverse event associated with “Covid” vaccination. It is admitted that most cases of pericarditis and myocarditis are reported in young adults who are vaccinated:
Fulminant myocarditis after the second dose of COVID‐19 mRNA vaccination
“Myocarditis is an adverse event associated with coronavirus disease 2019 (COVID‐19) mRNA vaccination. A 50‐year‐old man presented with dyspnea and resting chest pain after receiving the second dose of the COVID‐19 mRNA vaccine and developed cardiogenic shock. Fulminant myocarditis was diagnosed by endomyocardial biopsy and treated with intravenous corticosteroids.
Fulminant myocarditis is an adverse event associated with the COVID‐19 mRNA vaccination. Steroid pulse therapy may control the pathology of myocarditis caused by COVID‐19 mRNA vaccination.
Vaccination is an essential component of the public health strategy to end the coronavirus 2019 (COVID‐19) pandemic; 1 however, various vaccine‐associated adverse events are emerging as an issue that cannot be overlooked. Although the most frequent adverse events following COVID‐19 mRNA vaccine administration include fever, fatigue, headache, myalgia, and injection‐site pain; several serious adverse events, including myocarditis and pericarditis, have also been reported, particularly in adolescents and young adults. 2 , 3 In a recent database in Israel, the estimated incidence of myocarditis within 21 days after the second dose of BNT162b2 mRNA vaccination was 0.21 per 100,000 persons among male recipients aged>50 years. 4 Although most cases had mild clinical courses, there are a few reports of fulminant myocarditis with cardiogenic shock. 3 Treatment of myocarditis is tailor‐made, depending on the case, and often includes NSAIDs, steroids, intravenous immunoglobulin, and colchicine. 5”
“This case demonstrated that myocarditis, which is a relatively mild and curable complication of the COVID‐19 mRNA vaccine, may follow a fulminant course. Further, our case revealed that steroid pulse therapy may be useful for controlling the pathology of myocarditis potentially resulting from the COVID‐19 mRNA vaccination.”
Another report showed that the “viral” vector vaccines were associated with this same reaction when a middle-aged healthy female developed fulminant myocarditis after her first injection of the AstraZeneca vaccine. While this was a case in a female, the report reiterates that myocarditis is seen mostly in adolescent males. It also states that there is growing evidence of this reaction happening after vaccination:
Acute Fulminant Myocarditis After ChAdOx1 nCoV-19 Vaccine: A Case Report and Literature Review
“According to recent literatures, myocarditis is an uncommon side effect of mRNA vaccines against COVID-19. On the other hand, myocarditis after adenovirus based vaccine is rarely reported. Here we report a middle-aged healthy female who had acute fulminant perimyocarditis onset 2 days after the first dose of ChAdOx1 vaccine (AstraZeneca) without any other identified etiology. Detailed clinical presentation, serial ECGs, cardiac MRI, and laboratory data were included in the report. Possible mechanisms of acute myocarditis after adenoviral vaccine was reviewed and discussed. To our knowledge, a few cases of myocarditis after Ad26.COV2.S vaccine were reported, and this is the first case report after ChAdOx1 vaccine.
Growing evidence has shown that acute myocarditis is a rare complication after mRNA COVID-19 vaccinations, with an estimated incidence of ∼2 per 100,000 persons after BNT162b2 mRNA vaccine (1, 2), and the risk is higher in adolescent males. Typically, acute myocarditis occurs within 5 days after mRNA vaccination, and the mechanism is still unclear. Myocarditis after adenovirus or protein-based vaccines has seldom been reported. Here, we report the case of a 44-year-old female who had acute fulminant perimyocarditis following the first dose of ChAdOx1 nCoV-19 vaccine with no other identified etiology.”
The last case report is of a 27-year-old male who sadly succumbed to his illness. The authors state that, while most cases of myocarditis will resolve, fatal cases of fulminant myocarditis were being reported. They note that very few autopsies are performed on mRNA vaccinated individuals, hinting that this could be a bigger problem than reported. Interestingly, it is noted that this condition was observed in smallpox vaccination as well, something that was being used as a treatment in the recent “monkeypox outbreaks:”
Case Report An autopsy case report of fulminant myocarditis: Following mRNA COVID-19 vaccination
“The first mRNA vaccine for humans against the coronavirus disease 2019 (COVID-19) pandemic has been developed and is being used worldwide; however, postvaccination myocarditis has also been reported. Most reported cases of post-mRNA vaccination myocarditis are considered non-life-threatening [1,2]. However, there have been case reports of fatal fulminant myocarditis . Moreover, there are extremely few autopsy cases of myocarditis after mRNA vaccination .
“We report an autopsy case of a 27-year-old healthy man who presented with cardiopulmonary arrest 8 days after the first dose of the mRNA-1273 SARS-CoV-2 vaccine and was finally diagnosed with fulminant myocarditis. Mitral valve prolapse is relatively common in athletes, also known as arrhythmic mitral valve prolapse, and may present with ventricular arrhythmias and sudden cardiac death even in the absence of hemodynamic impairment [, , ]. In this case, endomyocardial biopsy performed before death showed no evidence of myocarditis; therefore, cardiopulmonary arrest due to arrhythmic mitral valve prolapse was considered a possible differential diagnosis. However, the autopsy showed obvious severe myocardial inflammation findings, leading to the diagnosis of myocarditis. It is difficult to distinguish hypertrophic cardiomyopathy from the athlete's heart occasionally , the patient presented with asymmetric left ventricular hypertrophy, but no findings characteristic of hypertrophic cardiomyopathy were seen on autopsy. Therefore, we considered that his left ventricular hypertrophy was a consequence of the athlete's heart and MR. Microscopic findings showed infiltration of T cells, macrophages, and eosinophils. Such myocarditis with lymphocytic and eosinophilic infiltration has been reported after mRNA COVID-19 vaccination [3,8]. Lymphocytic and eosinophilic infiltration was also observed in myocarditis after smallpox vaccination and a mechanism of immune-mediated myocarditis has been speculated . In addition, Frustaci et al. reported three cases of pathohistologically defined eosinophilic myocarditis after COVID19 vaccination, referring to the possibility of eosinophilic myocarditis (hypersensitivity myocarditis) as an allergic reaction and the possibility that immunosuppressive therapy including steroids may be effective .
Here, we reported an autopsy case of fulminant myocarditis following mRNA COVID-19 vaccination. The microscopic findings revealed that significant mixed inflammatory infiltration (T cells, macrophages, and eosinophils) and widespread fibrosis were observed in the left ventricle. Steroid therapy may be effective in the treatment of fulminant myocarditis following mRNA COVID-19 vaccination.”
Was Jacob's fulminant myocarditis a result of the vaccine? We most likely will never know. It is definitely plausible, given the association between the condition and the vaccines. Perhaps his episode had nothing to do with the vaccine and everything to do with the treatments that he was subjected to after his testing results came back? That seems to be the most likely case, given that Jacob was not expressing any severe symptoms at the time. It is clear that, just as in the case of the kids with the mysterious liver disease, the diagnosis of a mystery “virus” was the easy label to slap onto the case in order to stop investigation into other potential causes.
It is not possible to say with 100% certainty that either the liver disease experienced by the children or the fulminant myocarditis suffered by Jacob were direct results from the vaccines. These stories simply could have been floated out there in order to create the perception that these diseases are occurring in order to limit any noise generated from a possible increase observed with the use of the vaccines. Or they could be used to cover up any of the other possible treatments and toxicities we are exposed to on a daily basis. However, one thing that is for certain is that there was never any proven “viral” cause in any of these situations. In fact, they didn't even need any evidence of a “virus” at all in order to claim that a “virus” was the guilty party. It's a neat trick that virologists have given themselves where they can presume and declare a “virus” as a causative agent despite the contradictory findings and without ever having to prove their claim. The concept of the “virus” has always been used to stifle any investigation into causes of disease that will harm the interests of the industries that would lose profit from implicating their own products and/or practices. The mysterious “virus” is the perfect scapegoat to blame in order to keep faith in the the fraudulent theory, the inaccurate tests and the toxic treatments. It is the sleight of hand to keep people looking the other way.provided an uplifting update on Marvin Haberland's court case against virology. took a detailed look at the reason behind the outcome in the Haberland case. provided a great expose on why the toxic HIV drugs don't work. took a look at Reiner Fuellmich's "SARS-COV-2" journey to point out what he got right, and the most important thing he continues to get wrong.